How I Survived Melanoma Skin Cancer

How To Prevent Skin Cancer

How To Prevent Skin Cancer

Complete Guide to Preventing Skin Cancer. We all know enough to fear the name, just as we do the words tumor and malignant. But apart from that, most of us know very little at all about cancer, especially skin cancer in itself. If I were to ask you to tell me about skin cancer right now, what would you say? Apart from the fact that its a cancer on the skin, that is.

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How I Survived Malignant Melanom

By The Time You've Finished Reading How I Survived Melanoma Skin Cancer Seven Survivors Tell Their Stories. You'll Feel Like A New Person, with A New, More Positive Outlook! You will learn: 1. How do I know if I have melanoma? What are the signs and symptoms? I wanted to know why the doctor was so concerned when she looked at that little mole on my forearm. What was it that looked so sinister? How worried should I be? Was the doctor over-reacting? 2. What tests will the doctor carry out to see if I have melanoma? Will they be able to tell me on the spot if there is a problem? Or will I have to wait for days, fretting about whats going on? 3. How curable is melanoma? If they do tell me its melanoma, what exactly does that mean? Is it a death sentence? Will they tell me You have 12 months to live. Get your life in order and prepare for the worst.? 4. What are the stages of the disease? The reading Id done said that there were different stages of melanoma. What are the symptoms of each stage? What are the survival rates of each stage? If I had a later stage melanoma, wouldnt I know about it? Wouldnt I actually feel like I was sick? 5. How quickly does the disease progress or spread? Should I have gone to the doctor sooner? Id noticed the mole changing over about 3 months. Was this delay critical? 6. How is melanoma normally treated? Would I have to go through chemotherapy and radiation treatment? If so, for how long? What are the odds of curing the disease using these treatments? How extensive is any surgery likely to be? How big will the scars be? 7. What are the common side effects of the treatments? Would I lose my hair? Would I become sterile? What else could I expect? 8. What alternative treatments are available? Id heard of people going on special macro-biotic diets. Id seen lots of herbal remedies on the internet. Which of these are proven and documented, and which ones are snake oil? Is it possible to combine alternative treatments with surgical other western treatments? How do I find a doctor that is open to using both alternative and western treatments? 9. What are the latest treatments being developed, and who is carrying out clinical trials of these new treatments?

How I Survived Malignant Melanom Summary


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Author: Daryl Grant
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The writer has done a thorough research even about the obscure and minor details related to the subject area. And also facts weren’t just dumped, but presented in an interesting manner.

This ebook does what it says, and you can read all the claims at his official website. I highly recommend getting this book.

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AOhd Analogs In The Treatment Of Cancer

There is extensive literature available on the use of deltanoids as anticancer agents. With VDR being expressed in more than 30 nonclassical target tissues and many cancer-related cells, the deltanoids have been shown to inhibit cell proliferation in many tumor cell types in culture. Initial findings using hemato-poietic-derived tumor cells showed inhibition of cell proliferation. Further evidence indicated the versatile use of deltanoids across several nonhematopoietic malignancies including transformed breast, prostate, skin, lung, colon, ovary, pancreas cells, as well as neuroblastoma and melanoma cells.6675-7980 Although much of the mechanistic data gathered in vitro point toward cell cycle arrest, differentiation, and induction of apoptosis, the proposed mechanism of action of deltanoids is likely to differ based on the type of tumor model examined. No unified hypothesis has emerged so far on the basis for the anticancer effects of 1a,25-(OH)2D3and its analogs. In addition, as...

Use in Prevention and Therapy

Vitamin A is one of nature's primary anticancer substances, particularly in the skin and mucous membranes. Ample intakes of vitamin A have been shown to protect against cancers of the lung, bladder, prostate, larynx, esophagus, stomach, and colon. Vitamin A can prevent precancerous lesions, such as oral leukoplakia (white patches on the lips and mouth often found in smokers) and cervical dysplasia, from developing and may produce regression and disappearance of these disorders.15 As a cancer treatment, large doses of retinoic acid may reduce growth and recurrence of certain forms of skin cancer.16 As an antioxidant, beta-carotene helps provide protection against damage from many xenobiotics (such as polychlorinated biphenyls PCBs ). It may also reduce the risk of skin cancer associated with exposure to sunlight6 and radiation.2

Vitamin D And The Cancer Connection

The role of vitamin D in cancer prevention perhaps has been known for more than 50 years. Although excessive sun exposure has been documented to increase the risk of skin cancer, research conducted starting as early as 1936 has proved this population of patients with skin cancer to be at a lower risk for other types of cancer. Sun exposure has been correlated with decreased incidence of certain types of cancer such as cancers of the prostate, breast, and colon. Individuals residing in the U.S., which lies in the northern latitudes, have a risk for cancer incidence which is two to three times higher than the risk of cancer incidence of people living in sunnier, equatorial parts of the world.40 This intriguing observation by Apperly40 was followed by several epidemiological studies that demonstrated an inverse relationship between 25-(OH)D3 levels and cancer risk and

Anticarcinogenic Activity Of Natural And Synthetic Carotenoids

Cancer-preventive activity in interventional trials i.e., two Linxian trials (Linxian 1 and Linxian 2), the Alpha-Tocopherol Beta-Carotene (ATBC) cancer prevention study, the P-Carotene and Retinol Efficacy Trial (CARET), the Physicians' Health Study (PHS), and the Skin Cancer Prevention Study (SCPS). In addition to these studies, we have recently completed an intervention trial with supplementation of a mixture of natural carotenoids (lycopene, P-carotene, a-carotene, and others) plus a-tocopherol (Jinno, K., Nishino, H. et al., patent pending 2002-022958, 2002.1.31 see Section 4.3.8). In the CARET, the relative risk of lung cancer incidence was 1.3 in the group treated with P-carotene and retinal (p 0.02), 1.4 in the current smoker group treated with P-carotene and retinal, and 1.4 in the asbestos-exposed group treated with P-carotene and retinal. In the PHS, no significant modification in risk was found. In the SCPS, the relative risk for skin cancer was 1.4 in the smoker group...

Alternative Explanations for Low Cancer Risk in Vegetarians

Screening Bias For some forms of cancer, screening is effective in early detection. Specifically, some of the female cancers such as cervix and breast, as well as prostate cancer in men may be detected more frequently in populations that have a greater than normal degree of access to screening programs. For example, in the Adventist Health Study in which cancer incidence was monitored among SDA between 1976 and 1982, elevated incidence of skin cancer, prostate, and cervix cancers was observed.28 These findings may not be a reflection of an actual increased biologic risk of these forms of cancer in SDAs, but an artifact of more intense screening for cancers among SDAs than in the general population. There is a strong focus on healthy lifestyles among SDAs, as well as greater access to health services among SDAs than the general public. The same phenomenon may act in the opposite direction in regard to mortality, since screening most likely is associated with early detection and better...

Raw Evolutionary Diets Dont Work In Practice

there was a letter from Montrame which explained (their version) of the reasons and the course of events of Mrs. Burger's death. The letter explained that both Burgers acquired the habit of eating lots of meat during an experimental phase, after they and their friends discovered the beneficial effects of raw meats, particularly for people who suffered from cancers. After some time Mr. Burger developed one small cancer-like melanoma on his legs, which was removed by surgery. He stopped eating meat for some time, then. After some time, Mrs. Burger, who already was in menopause, did get her period again. The reason was cancer. But Mrs. Burger was addicted and couldn't break the habit of eating meat, she didn't believe in her husband's new theory that meat can also cause cancer. She ate it every day and after Orkos stopped delivering it to her, she ordered through other people.

Boring Statistics and Exciting Cases

Boring Routine Statistics America

There are 180 such patients in this study. Thirty different types of cancer are represented. While none of these are the ordinary skin cancers, 10 of them are the deadly malignant melanoma type of skin cancer. From 1974 through 1991, a total of 42 patients have died. Twenty-three of those patients (12.7 ) died from causes related to their cancer. There were 108 patients in the study representing 23 different types of cancer. No ordinary skin cancers were included, but 4 of the patients had malignant melanoma with metastases. This 62-year-old woman was first seen on 12 6 78. One month previously she had a mole removed from her back. This mole turned out to be a malignant melanoma. She had no radiation or chemotherapy. She was placed on a nutritional program. She is now 77 years old, quite well and quite active. She has had small a skin cancer removed from her face, but this was not melanoma and was unrelated to her previous disease. I bring this case to your attention because melanoma...

Production Of Phytoene In Mammalian Cells

Phytoene, which is detectable in human blood, was proved to suppress tumori-genesis in skin cancer models. It has been suggested that the antioxidative activity of phytoene may play an important role in its mechanism of action. To confirm the mechanism, more precise study should be carried out. However, phytoene becomes unstable when it is purified, and thus is very difficult to examine its biological activities. Therefore, stable production of phytoene within target cells was tried. As phytoene synthase encoding gene, crtB, has already been cloned from Erwinia uredovora,13 we used it for the expression of the enzyme in animal cells. Plasmids encoding phytoene synthase were transfected into NIH-3T3 cells and expression was determined by Northern blot. These cells expressed a

Effects of selenium on the skin immune system

The skin is the frontier of the immune system, the interface of the body with the external environment and the site of exposure of the immune system to muta-genic and oxidative damage from UV radiation (reviewed in Duthie ei al., 1999). Oxidizing agents are also produced by commensal microorganisms that reside on the skin. Se has been shown to have a vital role in protecting the skin from carcinogenesis and from oxidative damage (reviewed in McKenzie, 2000). In mice, dietary Se supplements or even topical application of Se significantly decrease the incidence of skin tumours, tumour size and mortality. Mice on Se-deficient diets also have significantly lower numbers of Langerhans cells in the skin, which may result from increased secretion of TNF-a, which triggers emigration of these dendritic cells. In humans, low Se intake has been correlated with increased incidence of skin cancer, but it is not clear yet whether Se supplements protect humans from skin damage and malignancy...

Dr George Schaller Year of the Gorilla

The benefits of Sunshine are improved by eating correctly as the Sun and fresh air act like magnets drawing toxic matter to the skin. When the body is clean and internally protected with beta-carotene you will be amazed at how long you can be in the Sun and how well you tan. When people eat correctly and are detoxified, exposure to the Sun cannot lead to skin cancer. A diet high in cooked fat (free radicals), chemicals, and low in green leaves has been positively linked to skin cancer. This is because free radicals and toxins in the unprotected skin are baked and mutated by the Sun's rays. Recently, researchers at Baylor College of Medicine found that people on a low-cooked-fat diet had a greatly reduced risk of developing pre-malignant growths and nonmelanoma skin cancers.

Antioxidant Activity

White tea has good antioxidative and antiaging functions . The oxidative stress in skin cells leads to immune system damage, and can promote skin cancer and photo damage, such as wrinkling or mottled pigmentation The white tea extract could limit and prevent the DNA damage in cells after exposure to sunlight. 22 Therefore, white tea extract is believed to be an effective antioxidative agent, and may provide antiaging benefits . 23


There is a lot of controversy about the sun today. There seems to be a total panic about getting any sunlight at all just in case you get skin-cancer. I feel that skin cancer doesn't result purely from over exposure to sunlight. But that damage to the skin from over exposure can be a trigger to cancer if you are unhealthy and your immune system is weak. The condition of your health and immune system is a result of diet and lifestyle.

Animal Studies

The direct influence of P-cryptoxanthin intake on the development of colon tumors has been studied in F344 rats (Narisawa et al., 1999 ). N-methyl-nitrosourea-induced colon cancer in rats is significantly reduced for those given high doses of P-cryptoxanthin (25 ppm dietary level). Twenty-four (96 ) of 25 control rats compared to only 17 (68 ) of 25 P-cryptoxanthin-supplemented rats developed tumors during the 30-week study (p 0.02). Lower doses of 1 or 5 ppm did not produce a statistically significant reduction in the number of tumors observed in similar study groups. Tissue levels were significantly elevated for the high-dose animals but not for those given the lower doses. Another rodent study (Nishino et al., 1999) investigated the effect of P-cryptoxanthin against tumorigenesis in mouse skin. Skin cancer was induced in 15 SENCAR mice, which were consuming P-cryptoxanthin at a level of 25 ppm in drinking water, by treatment with peroxynitrite and promotion with TPA...

Repairing the Damage

It is instructive to examine a condition called xeroderma pigmentosum to get an idea of how important this system really is. Because the cells of people afflicted with this disease lack certain DNA repair enzymes, any exposure to sunlight causes severe, chronic damage to skin leading to multiple skin cancers. In fact, these peoples' risk of skin cancer is two thousand times higher than people with normal repair mechanisms. Even the risk of internal cancers is increased twelve times higher than normal.501


There is a marked variation among countries in incidence of different cancers. Most of the variation in cancer risk among populations, and among individuals, is due to environmental factors, such as cigarette smoking and certain dietary patterns, that can affect one's risk of developing cancer. For example, individuals living in Australia have the highest worldwide lifetime risk of skin cancer, at over 20 percent, due to the high level of exposure to the sun of people in Australia. People in India have twenty-five times the average risk of developing oral cancer sometime during their lives due to the popularity of chewing tobacco in that country. In fact, India has the world's highest incidence of oral cancer, with 75,000 to 80,000 new cases a year. The population of Japan has the highest rates of stomach cancer in the world due to the high consumption of raw fish by the Japanese.

Causes of Cancer

Overweight and obesity are associated with increased risk of cancers of the breast, colon, endometrium, esophagus, kidney, and gallbladder. The following chemicals have been found to cause cancer coal tars and their derivatives, such as benzene some hydrocarbons aniline, a substance used to make dyes and asbestos. Radiation from a variety of sources, including the ultraviolet light from the sun, is known to lead to skin cancer. About 5 to 10 percent of cancers are hereditary, in that a faulty gene or damaged DNA that has been inherited predisposes a person to be at a very high risk of developing a particular cancer. Two genes, BRCA1 and BRCA2, have been found to cause some breast cancers. Other genes have been discovered that are associated with some cancers that run in families, such as cancers of the colon, rectum, kidney, ovary, esophagus, lymph nodes, skin melanoma, and pancreas.