Cure Crohns Disease Naturally

Breakthrough Crohns Disease Guide

In Breakthrough Crohn's Disease Guide, you'll learn how your digestive system really works absorbing nutrients and fluids from the foods you eat while compacting and sending waste products along their way. You'll discover how your own immune system, in trying to fight a perceived threat, has sent white blood cells to the smooth lining of your digestive tract where they do their best to root out infection.

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No More Crohn's Disease

No More Crohn's Disease is a product of Cathy Rubert's personal research and many years of trial and error. This book reveals Cathys powerful 4-step plan against Chron's disease. Inside her book you will discover how to quickly and easily implement the 4-main all-natural steps that will immediately start combating Crohn's Disease (put the simple steps into action and Crohn's Disease will become a problem from the past!) Time to be freed from the pain and discomfort caused by Chron's disease. With the help of Cathy Rubert's No More Crohn's Disease, you can now enjoy an effective cure without the costly trips to the doctor. You will also be safe from any side effects because the entire program is natural.

No More Crohns Disease Summary

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Cured My Crohns

If you've ever gotten the fateful diagnosis you've got Crohns, you will know the massive upset that it can have on your way of life and how you feel about yourself and your relationship to other people. If you talk to your doctor about natural diets or some other method of curing your Crohns disease they will tell you that there is no way to fix it. However, there is often more to the story than modern medicine will tell you. New Age medicine is not a bunch of nonsense that hokey people subscribe to; New Age medicine fills in the gaps of knowledge that we have with modern medicine and helps us understand what is going on with our bodies. You will learn how to cure Crohns from someone who has cured it himself and has lived for over 10 years completely free of disease!

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Inflammatory Bowel Disease Ulcerative Colitis and Crohns Disease

There are two major forms of chronic inflammatory bowel disease (IBD). Ulcerative colitis is an ulcerative disorder of the mucosa of the colon, whereas Crohn's disease is characterized by transmural inflammation, most often in the small intestine. Both produce abdominal pain and diarrhea, which can be bloody. iBd tends to wax and wane, with periods of intense active disease followed by long periods of remission. Although the cause is not clear, IBD appears to be an autoimmune reaction, in which overzealous immune cells attack the tissues of the intestinal wall. for hospital care and intestinal surgery. Food sensitivities may aggravate IBD, and identification and avoidance of offending foods may increase chances of remission.14 During acute exacerbations of Crohn's disease, enteral nutrition with protein hydrolysate diets is effective and reduces need for steroid therapy.15

Crohns disease

Crohn Disease Children

Although Crohn's disease (CD) most commonly affects the terminal ileum and or the colon, involvement of the upper gastrointestinal tract is Figure 7.2 Small pre-pyloric ulcers (arrows) in a 14-year-old girl with Crohn's disease. Figure 7.2 Small pre-pyloric ulcers (arrows) in a 14-year-old girl with Crohn's disease. Systemic disease Crohn's disease

Criteria for histological diagnosis

Riddell stated that to differentiate indeterminate colitis lesions from Crohn's disease lesions such as submucosal and subserosal lymphoid aggregates away from areas of ulceration, non-necrotizing granulomas and skip areas should be absent.16 This is particularly true where there is nonspecific ileal involvement or gastritis and special stains for Helibacter pylori are negative. The most frequently used diagnostic study for distinguishing indeterminate colitis from Crohn's disease (in addition to ileocolonoscopy with biopsy) is the small-bowel X-ray. Although the above criteria would appear to allow differentiation between Crohn's disease and ulcerative colitis on the basis of pathology, Farmer et al documented disparity among pathologists reviewing cases of colonic IBD.17 The diagnoses of gastrointestinal pathologists differed from that of the referring institution in 45 of surgical specimens and 54 of biopsy specimens. Of 70 cases initially diagnosed with ulcerative colitis, 30 (43...

Serologic markers and defining inflammatory bowel disease categories

We are currently evaluating the role of perinuclear anti-neutrophil cytoplasmic antibodies (PANCA) and anti-Saccharomyces cerevisiae antibodies (ASCA) in identifying patients with ulcerative colitis and Crohn's disease previously diagnosed with indeterminate colitis.4 While PANCA was positive in 68 of those favoring ulcerative colitis, and ASCA was positive in 37 of those favoring Crohn's disease, 86 of our patients with indeterminate colitis were both p-ANCA and ASCA negative. Recently, Joossens et al correlated serological markers with prospective follow-up evaluation in 97 adult patients with indeterminate colitis.8 After a mean follow-up of 6 years, 32 of the adult patients with indeterminate colitis were reclassi-fied as having ulcerative colitis or Crohn's disease, half of whom were positive for p-ANCA or ASCA. However, almost half of the patients with indeterminate colitis (48.5 ) remained p-ANCA ASCA negative and continued to have characteristics of

Epidemiological aspects of indeterminate colitis

Most children with indeterminate colitis are evaluated because of recurrent abdominal pain and diarrhea, with a smaller number noting hema-tochezia.12 In young children (less than 5 years of age), failure to thrive is more prominent than seen in ulcerative colitis.13 Hassan et al noted no difference in gender, age at diagnosis or types of symptom at presentation among the 38 newly diagnosed children with ulcerative colitis, indeterminate colitis and Crohn's disease.14 'Favor Crohn's disease' 18 - except for findings of No small-bowel X-ray features of Crohn's disease No distinguishing features 39 Crohn's disease including areas of focal colitis, focal gastric or duodenal inflammation, anal fissures or isolated granulomas adjacent to ruptured crypts. Features 'favoring Crohn's disease' were present in 20.4 of children with indeterminate colitis, none of whom had granulomas, radiological evidence of small-bowel Crohn's disease or perianal findings. Endoscopic and histological findings...

ILactosefree cows milk proteinbased

Lactose-free cow's milk protein-based formulas contain the same ingredients as other infant formulas based on cow's milk, except that glucose polymers from corn syrup solids are substituted for lactose. They may be useful during periods of secondary disaccharidase deficiency due to acute enteritis or chronic conditions affecting the integrity of the small intestine such as diarrhea, enteropathies and Crohn's disease.

Gastrointestinal function and disease

Two major disorders, Crohn's disease and ulcerative colitis, are conventionally grouped together under the heading inflammatory bowel disease because both are characterized by chronic inflammation in the gut. However, it is best to consider them as separate conditions because they have characteristically different pathology, clinical courses, complications and management. The aetiology of neither is known. 5.5.1. Crohn's disease. Crohn's disease may affect any part of the gut from mouth to anus. Characteristically it occurs in the ileocaecal region and colon, and the inflammation is patchy or discontinuous. It frequently recurs after surgical resection of the affected areas of gut at or near the point of anastomosis of the bowel. The involved intestine is thickened, with ulceration of the mucosa, stricturing and fistula formation. Mouth ulcers and perianal abscesses are characteristic. Histologically there is transmural inflammation, with mononuclear cells, lymphoid aggregates and...

Structure and Use of this Book

Chapter 4, Food Classification, is based on a variety of source texts and applies their concepts to the most common Western foods. Here you will find detailed information about the nature and use of foods and food groups in nutritional therapy. In case of contradictions between the authors regarding classification, I endeavored to classify foods according to my own clinical experience. The clinical examples in Chapter 5, Analogy of Western Diagnoses with Syndromes in Traditional Chinese Medicine (TCM), intend to establish a bridge to Chinese syndrome diagnosis. By their very nature, these offer abridged and incomplete introductions to a very large field of knowledge. They are designed to encourage Western practitioners to combine Western with Eastern thinking. Lay readers are advised that for disorders such as hypertension, asthma, Crohn disease, etc., any therapeutic measure should always be discussed

Drinking Water For Special Health Needs

Some people are more vulnerable to microbial contaminants such as Cryptosporidium (or crypto), which isn't destroyed by chlorination. More often found in surface water than ground water, crypto may cause nausea, diarrhea, or stomach cramps when healthy people ingest it. For people who are more vulnerable, the symptoms may be more severe and perhaps life-threatening. That includes people with HIV AIDS or other immune system disorders (such as lupus or Crohn's disease), organ transplants, the elderly, children, and those undergoing chemotherapy.

Overview and Conclusions

Other potential uses of immunoregulatory probiotics (e.g. in controlling inflammatory diseases at the gut surface) have only recently begun to attract research attention (Venturi et al., 1999), partly because the microbial gut mucosal signalling mechanisms are only beginning to be understood by microbiological researchers (Haller et al., 2000). A recent pilot study (Gupta et al., 2000) showed promising preliminary results for the use of L. rhamnosus GG as a dietary supplement to reduce clinical indices of GI-tract inflammation in children with Crohn's disease. As research starts to define the interactions of the gut microflora and the immune system in the maintenance of health, so it is likely that new avenues for dietary intervention will become the focus of research efforts.

Clinical signssymptoms

Recent evidence has shown that GERD affects the quality of life significantly in adults, and probably also in children (and their parents), although quality of life is more difficult to evaluate in infants and young children. The developing nervous system of infants exposed to acid seems susceptible to pain hypersensitivity despite the absence of tissue damage.70 The role of hypersensitivity to dietary allergens, both in exclusively breast-fed and formula-fed infants, is likely to be underestimated at present. The 'hygiene hypothesis' suggests that the Th2-predominant immune response at birth in the industrialized world is insufficiently skewed towards a well-balanced Th1 Th2 response.71 Lack of controlled chronic or repetitive inflammation of the mucosa during the first months of life may account for the dramatic increase in atopic disease during infancy and childhood (which is a Th2 response) and the increase in autoimmune diseases such as diabetes and Crohn's disease in adolescents...

Food allergies classification

This reaction is essentially mediated by T lymphocytes, with tissue damage also caused by macrophages responding to T-cell cytokines. The pattern of T-cell responses (Th1 or Th2) may determine overall immunopathology. The classic type IV reaction is a Th1 response, as in Crohn's disease, while both Th1 and Th2 reactions occur in food allergy.

Diagnosis and differential

Coliti Plesiomonas

Ulcerative colitis Crohn's disease Henoch-Sch nlein purpura Hemolytic uremic syndrome Intestinal ischemia Intussusception The diagnosis of ulcerative colitis is established by the information gathered from a detailed symptom and family history, physical examination, and a combination of laboratory, radiological, endoscopic and histological findings. It is important to exclude other etiologies, such as an infectious process, and to distinguish ulcerative colitis from Crohn's disease. Colonic inflammation is typically characterized by bloody diarrhea with abdominal cramping. The differential diagnosis of colitis depends upon the age of the child at the time of evaluation. In infancy, necrotizing enterocolitis, Hirschsprung's enterocolitis and allergic colitis are common. In contrast, in the older child and adolescent, enteric infection and IBD are the most common diagnoses. Causes of colitis are listed in Table 25.3. In patients with painless rectal bleeding, other conditions (Meckel's...

Interactions with other nutrients and drugs

The synergistic effects of ro-3 and ro-6 fatty acids and drugs used in the treatment of Crohn's disease have been reported.6566 Amino-salicylic acid (5-ASA) therapy with ro-3 fatty acids supplementation in the treatment of pediatric patients with Crohn's disease delayed the relapse of episodes. The synergistic effect of the drug and supplement were observed by 5-ASA inhibition of factors of the inflammatory cascade cyclooxygenase, thromboxane-synthetase, and platelet activating factor (PAF) synthetase , production of IL-1 and free radicals, antioxidant activity, and ro-3 fatty acid inhibition of the PAF synthetase.65 Omega-3 but not ro-6 fatty acids have shown immunomodulatory properties and an increase in pro-inflamatory cytokines in patients receiving enteral ro-3 and ro-6 fatty acids used as adjuvant therapy to corticosteroid medication for the treatment of Crohn's disease.66 Omega-3 fatty acids have been shown to decrease the nephrotoxicity caused by cyclosporine therapy used to...

Disorders of the enteric nervous system

A number of patients have been reported with different non-infectious inflammatory diseases. Clearly, classical mucosal inflammatory conditions such as Crohn's disease and necrotizing enterocolitis may result in damage to both enteric nerves and muscular structures of the bowel. However, other autoimmune diseases affecting the gut, such as celiac disease and ulcerative colitis, may also result in severe dysmotility. It is becoming apparent that the neuromusculature of the bowel may become involved in the inflammatory process.

Prognosis

Crohn's disease is a chronic inflammatory process that cannot be cured by current medical therapy or by resection. Nevertheless, children with Crohn's disease can manage their disease and achieve their goals. General Eisenhower had Crohn's disease and he was elected President of the United States. Athletes with Crohn's disease perform effectively and children should be encouraged to pursue their dreams. Education, recognition of early manifestations of disease and compliance will enable chil

Epidemiology

A recent systematic review by Loftus et al3 analyzed the epidemiology and natural history of Crohn's disease in population-based cohorts from North America. The prevalence rates of Crohn's disease ranged from 26.0 to 198.5 cases per 100 000 persons. The incidence rates ranged from 3.1 to 14.6 cases per 100 000 person-years. Based on an estimate of 300 million people in North America, there are approximately 400 000 to 600 000 patients living with Crohn's disease and approximately 9000 to 44 000 people are newly diagnosed in North America with Crohn's disease each year.3 There appears to be a slight female predominance, with the percentage of females with Crohn's disease ranging from 48 to 66 .3 Africa and southern Europe.4-9 This geographic difference is also observed within individual countries including the USA.10 Despite the low incidence and prevalence of IBD in Africa,11 similar rates for Crohn's disease have been observed between African-American and Caucasian people.12 Both...

Radiological imaging

In a further attempt to distinguish between the various forms of pediatric IBD, 99m-technetium white cell scans and magnetic resonance imaging have been evaluated in children with indeterminate colitis, ulcerative colitis and Crohn's disease.18-20 The technetium scan had a sensitivity of only 76 compared with colonoscopy and biopsy.19 Others found that conventional magnetic resonance imaging had a low sensitivity (40 ) for detecting Crohn's disease and did not correlate with the severity of inflammation.20 However, gadolinium-enhanced magnetic resonance imaging in combination with oral polyethylene glycol (PEG) solution (used to distend the small bowel) may be more discriminating.21 With the latter technique, increased wall thickness was noted in 26 26 children with Crohn's disease while those with indeterminate colitis and ulcerative colitis showed mild parietal contrast enhancement but not bowel wall thickening. Further confirmation of these observations is needed.

Natural history

With time, 50-72 of adult patients and 64 of pediatric patients with indeterminate colitis can be reclassified as having definite ulcerative colitis or Crohn's disease during subsequent obser-vation3-4,9-11,22 (Table 24.3). After a mean follow-up period of 14 months, one group reported that 33 of 36 patients with indeterminate colitis were reclassified as ulcerative colitis and 17 as Crohn's disease.9 Meucci et al reported that 37 of 50 patients (74 ) changed from indeterminate colitis to a definite diagnosis of ulcerative colitis or Crohn's disease during follow-up with a cumulative probability of 80 within 8 years of diagnosis.10 In contrast, Wells et al followed 16 patients with indeterminate colitis for a mean of 10 years and observed that three were reclassified with ulcerative colitis, one with Crohn's disease and the rest remained indeterminate.11 The course of indeterminate colitis in 36 Swedish children after a mean follow-up of 4.6 years was analyzed by Hildebrand et al.3...

Diagnosis

Both single- and double-contrast barium studies show poor sensitivity in documenting gastritis and gastropathy, although in some conditions they may be helpful. Double-contrast studies of the upper gastrointestinal tract can reveal an irregular mucosal profile, nodularity, ulceration, lumen narrowing or mass effect in almost 50 of children with Crohn's disease.62 A double-contrast upper gastrointestinal tract series in varioliform gastritis revealed multiple, well-defined, circular filling

Complications

Weight loss and malnutrition are the most prevalent nutritional disturbances in patients with IDB.103 Approximately 85 of children with Crohn's disease have a history of weight loss at initial diagnosis.127 Malnutrition is mainly due to decreased intake caused by either primary anorexia from proinflammatory cytokines, or intestinal inflammation. An active inflammatory process, especially associated with fever, may further increase the body's caloric consumption. Estimates suggest that an increase in 1 C increases the metabolic rate by 7 . For this reason, children with Crohn's disease who experience recurrent febrile episodes often need to receive additional calories. As the result of the location of chronic intestinal inflammation in Crohn's disease, a variety of micronutrient deficiencies may occur. These deficiencies include water-soluble vitamins (mainly folic acid and vitamin B12), fat-soluble vitamin D, and minerals (iron, copper and zinc).103 Deficiency of these micronutrients...

Treatment

Management of children with Crohn's disease includes medical therapy, potential surgical intervention as well as nutritional and psychiatric support. Education of the patient and the family is a critical aspect of care that should not be overlooked. The IBD Notebook that is available through the North American Society for Pediatric Gastroenterology, Hepatology and Nutrition is a valuable resource empowering patients to understand and control their disease. The general guiding principles for treatment are to control inflammation and achieve normal growth while maintaining a high quality of life. For patients with mild-to-moderate disease, initial therapy with 5-aminosalicylic acid (5-ASA) and antibiotics may induce remission. For patients with more active disease, corticosteroids may provide a more rapid response. Initial treatment with 6-mercaptopurine seems to decrease steroid dependency, but most clinicians reserve immunomodulator therapy for patients who fail to maintain a...

Enteric myositis

In Crohn's disease, infiltration of the smooth muscle layer with lymphocytes has been found, but no clue as to whether this is part of the Crohn's disease or an autoimmune phenomenon. Acquired myositis has been described in children in a number of reports. Two have presented with functional intestinal obstruction at 1 and 2.5 years of age. In these patients a dense lymphocyte infiltrate mainly of T cells was found along the large and small intestine. Both responded to immuno-suppressive treatment. A further child with similar histological findings but in whom there was clear evidence of an autoimmune process has more recently been described.8 In this patient features were noted that suggested that the intestinal muscle cells themselves were taking part in the

Medical therapy

The observation that the majority of patients with indeterminate colitis over time are reclassi-fied as ulcerative colitis or Crohn's disease makes it difficult to know whether indeterminate colitis represents a separate form of IBD. Perhaps because of the small number of patients with indeterminate colitis, the response to various drug regimens in this population has not been specifically addressed.7 In our program the choice of therapeutic intervention is selected depending on the severity of symptoms, extent and severity of endoscopic and histological findings and laboratory parameters23 (Table 24.4). For most patients, drug therapy is similar to that indicated for patients with ulcerative colitis of comparable extent and severity. These include 5-aminosalicylic acid (5-ASA) preparations for mild disease and corticosteroids and immunomodulatory therapy for moderate and severe disease. However, we are more likely to use metronidazole in this population, especially where there is...

Followup management

Commonly used antibiotics for Crohn's disease prevent development of osteopenia and osteoporosis, regular bone density study using DEXA is recommended for patients on prolonged corticosteroid therapy. There are no proven therapeutic interventions in children with Crohn's disease that have been shown to prevent recurrence of disease or maintain remission. Nevertheless, immunomodulator therapy and 5-ASA are often used in this role. Table 23.13 Pediatric Crohn's Disease Activity Index (PCDAI) (adapted from reference 194)

Surgical treatment

Ulcerative colitis (58 ) and Crohn's disease (72 ). However, this latter group observed a greater frequency of fistulae after IPAA in patients with indeterminate colitis (26 ) versus for ulcerative colitis (10 ).35 Post-IPAA complications resulting in pouch removal were higher for indeterminate colitis (19-28 ) versus ulcerative colitis (0.4-8 ) in some studies32,33 but not in others.35-37 It is our practice to repeat colonoscopy, usually with concurrent EGD, during selected periods of relapse to assess whether histological changes consistent with ulcerative colitis or Crohn's disease have developed. This is especially the case if colectomy and IPAA are being considered because of refractory disease. At this time, repeat small-bowel X-ray is usually obtained so that patients with ileal Crohn's disease would be excluded and patients with persistent indeterminate colitis would be counseled regarding the potentially greater risk of pouch complications. Often, patients with indeterminate...

Pathophysiology

Data from studies on tissue samples from patients with IBD and experimental animal models of colitis have demonstrated unbalanced Th1 vs. Th2 responses in the intestinal mucosa.84,104,105 The mucosa of patients with established Crohn's disease is dominated by CD4+ lymphocytes with a Th1 phenotype, characterized by the production of interferon-y and interleukin (IL)-2 2 whereas the mucosa in patients with ulcerative colitis may be dominated by CD4+ lymphocytes with an atypical Th2 phenotype, characterized by the production of transforming growth factor (TGF)-P and IL-5, but not IL-4.2 CD4+ lymphocytes clearly play an important role in the pathogenesis of tissue damage in inflammatory bowel disease, particularly in Crohn's disease.84,106 Patients with disorders such as glycogen storage disease 1b, which are associated with abnormalities in neutrophil function may exhibit clinical manifestations of IBD. These observations suggest that the mechanisms leading to mucosal injury are not...

Vitamins Fat Soluble

Because they dissolve in fat, vitamins A, D, E, and K are called fat-soluble vitamins. They are absorbed from the small intestines, along with dietary fat, which is why fat malabsorption resulting from various diseases (e.g., cystic fibrosis, ulcerative colitis, Crohn's disease) is associated with poor absorption of these vitamins. Fat-soluble vitamins are primarily stored in the liver and adipose tissues. With the exception of vitamin K, fat-soluble vitamins are generally excreted more slowly than water-soluble vitamins, and vitamins A and D can accumulate and cause toxic effects in the body.

Human Studies

In a study of Crohn's disease, a chronic intestinal inflammatory condition, a group of 28 patients was compared to a control group of 23 healthy subjects. In the study, Crohn's disease was found to correlate with statistically lower levels of lutein, zeaxanthin, a-, and P-carotene, but not P-cryptoxanthin (Rumi et al., 2000). In cord blood samples obtained from 96 infants it was observed that P-carotene levels, but not P-cryptoxanthin or retinol, were lowered by maternal smoking (Moji et al., 1995). Among breast cancer patients (n 266) studied in India, lowered levels of

Menetriers disease

Upper gastrointestinal X-ray series identifies the characteristic gastric rugae hypertrophy, predominantly in the fundus and in the corpus of the stomach. Thickened gastric mucosal folds have also been demonstrated by additional diagnostic imaging modalities, such as ultrasound and computed axial tomography. Endoscopy and histology should be performed in all patients to confirm the diagnosis, to search for the pathogen and to exclude different diagnoses, such as eosinophilic gastritis, primary gastric lymphoma, gastric carcinoma, Crohn's disease of the stomach and other conditions in which hypertrophic gastric rugae occur. Endoscopy shows swollen gastric folds predominantly in the greater curvature of the stomach or within the entire body region of the stomach, large quantities of gelatinous mucus and, less commonly, multiple areas of focal erosions in the fundus. Histology usually shows considerable elongation and complexity of the superficial epithelium, so-called foveolar...