New Autoimmune Diseases Cure

The Autoimmune Bible System

The Autoimmune Bible system was developed by Mark Steve, who was suffering from an autoimmune disease that is not treatable. He tried other medicine and did not work, but after turning to the Bible, he was able to get the best healing for the disease. The product discusses in detail the Bible recipes and also the ingredients which come with it, which can be used to fight the diseases. God has provided the people with healing through the readily available things which can be found in the grocery, and it only needs one to follow them strictly, and you will get healed. The Bible has specific herbs, which it has mentioned, which could be used to heal the various disease which we are suffering which include the autoimmune disease and diabetes which continue to be a problem to many. The program can be accessed through the e-book, and some bonuses come with the program and maximum benefits from the program. You don't need any technical skills or other intermediate skills to use the product since it entails using the Bible. Continue reading...

The Autoimmune Bible System Summary


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Author: Mark Steve
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The Autoimmunity Bible & Norton Protocol

A former autoimmunity (lupus) sufferer and alternative health specialist teaches you how to: Eradicate your autoimmune disease by addressing the single most important chemical imbalance on the cellular level as per groundbreaking new discoverie in the field. Avoid the traps that your adrenal glands set for you once you start using steroids. This reaction has been found to be responsible for autoimmunity flares in 81% of cases according to recent studies. If your disease caused a rash, now you can clear it out by getting rid of it's underlying triggers. Gain instant relief within 4 days to a week. Eliminate the debilitating life-altering pain in your joints and re-claim your life from the claws of autoimmunity. Eliminate the 2 overlooked obstacles that make any healing agent futile if your are suffering from an autoimmune disease. This is exactly why no drugs can cure autoimmune disorders. It's like pouring water into a hollow bucket. Reverse the processes that are most likely scavenging your kidneys. Continue reading...

The Autoimmunity Bible & Norton Protocol Summary

Contents: Ebook
Author: Julia Liu
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Price: $37.00

Gastritis associated with other autoimmune disease

Gastritis with or without atrophy has been described in association with several autoimmune diseases, such as connective tissue disorders, autoimmune thyroiditis and vitiligo. Histological gastritis has been reported in 25 of 27 diabetic children undergoing gastroscopy for upper gastrointestinal symptoms only half of them had evidence of macroscopic involvement of the gastric mucosa, including erosion and ulcers.60 A teenage girl with scleroderma and atrophic gastritis has been reported.3

The Aging Immune System

This means that we ran a higher risk of cancer of blood-forming tissues, such as leukemia, myeloma, and lymphoma, as well as immune incompetence. Such a situation leaves the body unable to protect itself from even common infections and increases risk for autoimmune diseases. As with so many other health-related disorders, the incidence of immune-related diseases in the population appears to be increasing and many can be correlated directly to poor nutritional practices in all age groups. Because immune overreaction is common with aging, autoimmunity due to other factors, such as viral infections and environmental toxins, can be worse in the aged. Interestingly, autoimmunity to brain proteins is much higher in cases of Alzheimer's, Parkinson's, and Lou Gehrig's diseases (ALS).512

Clinical signssymptoms

Recent evidence has shown that GERD affects the quality of life significantly in adults, and probably also in children (and their parents), although quality of life is more difficult to evaluate in infants and young children. The developing nervous system of infants exposed to acid seems susceptible to pain hypersensitivity despite the absence of tissue damage.70 The role of hypersensitivity to dietary allergens, both in exclusively breast-fed and formula-fed infants, is likely to be underestimated at present. The 'hygiene hypothesis' suggests that the Th2-predominant immune response at birth in the industrialized world is insufficiently skewed towards a well-balanced Th1 Th2 response.71 Lack of controlled chronic or repetitive inflammation of the mucosa during the first months of life may account for the dramatic increase in atopic disease during infancy and childhood (which is a Th2 response) and the increase in autoimmune diseases such as diabetes and Crohn's disease in adolescents...

Mercury and Multiple Sclerosis

Based on compelling evidence, the consensus in the medical community is that MS is an autoimmune disease. The big question is what triggers this autoimmunity There is growing evidence that a chronic viral infection (Herpes virus 7) plays a major role in this disorder, yet we also know that environmental and nutritional conditions can alter the course of the disease. High-fat diets, low in omega-3 fatty acids (from seafood) and high in omega-6 fatty acids (from vegetable oils), promote the disease and increase the severity of complications. Likewise, high antioxidant intake combined with a low-fat diet and increased DHA intake can reduce the severity of MS.

Early Problems with Vaccines

We now recognize that many viruses can adversely affect the immune system, either by depressing its function or causing it mistakenly to attack cells and tissues of the person exposed to the virus. The disorder is called autoimmunity, and the process, molecular mimicry. The measles virus is notorious for suppressing the immune system. Not uncommonly, it will also trigger autoimmune reactions. One of the most devastating autoimmune reactions to measles is called subacute sclerosing panencephalitis, a highly fatal disorder of young children.

Evaluation of Human Immune Response

Nutrition research offers a very interesting and potentially novel way to study the human immune system, and provides an important counterpart to the study of the immune response in primary or secondary immune deficiency where infection, autoimmunity or malignancy are manifest at clinical presentation. While it is clear that there is substantial variation in the normal immune response, the basis of this difference, whether genetic or environmental, remains to be determined. Fundamental studies are needed to determine how nutrient status may influence the development and expression of host genes involved in the immune response. Bendich (1995) has proposed that tests of immune function should be considered in determining the recommended daily allowance (RDA) of certain nutrients, since the levels of several micronutrients needed to support optimal immune function are often higher than those levels needed to qualify as clinical nutrient deficiency, which are usually defined in...

Tissue damage by oxygen radicals

Radicals may interact with any compounds present in the cell, and the result may be initiation of cancer, heritable mutations, atherosclerosis and coronary heart disease or autoimmune disease. The most important, and potentially damaging, such interactions are 2 With individual amino acids in proteins. This results in a chemical modification of the protein, which may therefore be recognized as foreign by the immune system, leading to the production of antibodies against the modified protein that will also react with the normal, unmodified body protein. This may be an important factor in the development of autoimmune disease.

Prevention and nutrition

Low-dose aspirin was shown to have small to moderate benefits for prevention of preeclampsia within certain groups of women. A Cochrane analysis demonstrated that in women at increased risk for preeclampsia, 69 women would need to be treated with low-dose aspirin to prevent one case of preeclampsia 27 . However, in the subgroup of women at highest risk for preeclampsia (because of histories of previous severe preeclampsia, diabetes, chronic hypertension, renal disease, or autoimmune disease), only 18 would need to be treated with low-dose aspirin to prevent one case of preeclampsia 27 .

What Is Celiac Disease

Celiac disease, also known as celiac sprue and gluten intolerance, is a genetically based autoimmune disease characterized by sensitivity to proteins found in the cereal grains wheat, barley, and rye. Having a genetically based disease means that the potential to develop the disease was inherited from one or both parents. Having an autoimmune disease means that the body's own immune system damages tissues of the body when presented with something it views as harmful. In the case of celiac disease, the body views certain proteins found in wheat, barley, and rye as harmful.

Putative involvement of lymphoepithelial interactions

Animal studies have suggested differential effects of antigen dose and feeding frequency on the mechanisms of tolerance induction (Brandtzaeg, 1998). At very high doses, both Th1 and Th2 cells were shown to be deleted following initial activation, an event apparently depending on apoptosis in Peyer's patches (Chen et al., 1995). Anergy and clonal deletion would be antigen-specific events, in contrast to active suppression resulting from deviation of cytokine profiles induced by T-cell stimulation locally or in regional lymph nodes or the liver (Knolle et al., 1999 Limmer et al., 2000) after distant transport of antigen in APCs or epithelial exosomes (Fig. 14.5). Experiments performed to induce therapeutic tolerance via the gut in various autoimmune disease models have relied on a bystander effect of stimulated T-cells, which, through immune deviation, have preferentially secreted down-regulatory cytokines, particularly TGF-p (Weiner et al., 1994). It has been suggested that the gut...

Digestion and Absorption

Protein Digestion Dipeptide Tripeptide

Entire proteins are sometimes absorbed, too, albeit in very small amounts. Since their sequences do not correspond to any of the bodies' own proteins, they are recognized as foreign by immune-competent cells. Physiologically, the purpose of this process may be to stimulate intestinal IgA and IgG secretion, thereby maintaining an important defense mechanism. There is also discussion, however, whether increased intestinal permeability (e. g., leaky gut syndrome in neonates) might be responsible for food allergies and autoimmune diseases.

Disorders of the enteric nervous system

Chronic inflammation and autoimmune disease A number of patients have been reported with different non-infectious inflammatory diseases. Clearly, classical mucosal inflammatory conditions such as Crohn's disease and necrotizing enterocolitis may result in damage to both enteric nerves and muscular structures of the bowel. However, other autoimmune diseases affecting the gut, such as celiac disease and ulcerative colitis, may also result in severe dysmotility. It is becoming apparent that the neuromusculature of the bowel may become involved in the inflammatory process.

Enteric myositis

A similar inflammatory cell infiltrate was described in 12 of 27 Bantu children from South Africa who had some sporadic form of an acquired degenerative enteric myopathy (V.V. Smith, personal communication). However, no information was given as to whether the children suffered from other autoimmune diseases or had autoantibodies present. Nor was there information about response to immuno-suppressive treatment.


The immune system has evolved to combat the constant threat of tissue invasion by microorganisms. If, however, the immune system is directed against innocuous antigens or tissue antigens, the same immune responses that are vital for defence against microorganisms can result in autoimmune disease and allergy. The adaptive immune response is reliant on the properties of B- and T-cells that enable the response to be powerful, flexible and antigen-specific and exhibit immunological memory. B-cells secrete antibodies that are effective against extracellular bacteria and their toxins, whereas CD8 T-cells are adept at neutralizing virally infected cells. CD4 T-cells, also known as Th-cells, do not directly neutralize invading pathogens instead, they interact with other cells (e.g. macrophages and B-cells) to direct a coordinated, antigen-specific immune response against microorganisms. CD4 Th-cell differentiation can be usefully considered to be either Th1- or Th2-biased. Th1-biased immune...

Vitamin A

With aging we also see a decrease in an important immune chemical called interleukin-2, which affords protection by enhancing T-lymphocyte production and function. Carotenoids improve age-related declines in interleukin-2 production. Another cytokine, interleukin-6, increases with aging519 and is associated with autoimmune diseases, Alzheimer's disease, and cancer.520


Initially, it seemed that differentiation of type 2 diabetes from type 1 diabetes and MODY would be straightforward. Children with type 1 diabetes often presented emer-gently at an early age with ketoacidosis, and the great majority had evidence of islet autoimmunity, with seropositivity to pancreatic islet cells and islet antigens, including glutamic acid decarboxylase (GAD65), tyrosine phosphatase IA-2, and insulin. In contrast, adults with type 2 diabetes had a more protracted course, mild symptomatology, and no evidence of islet autoimmunity. Children and adults with MODY were also mildly symptomatic but had a strong family history of early-onset disease. adolescents with type 1 diabetes are obese, and a minority (particularly Asians) may fail to show evidence of islet autoimmunity 1 . Most confusing is the evidence of islet autoimmunity in a significant percentage of children and adolescents with type 2 diabetes. Like children with type 1 diabetes, children and adolescents with...

Type Diabetes

In addition to the genetic contributions of type 1 diabetes, it is becoming evident that additional factors, such as environmental influences, are also involved in the development of the disease. Such factors include viruses, such as enteroviruses, rotavirus, and rubella (257,258). Nevertheless, even though Finland has effectively eradicated rubella through vaccination, it has one of the highest incidences of type 1 diabetes. This therefore supports the hygiene hypothesis, which proposes that environmental exposure to microbes early in life promotes innate immune responses that suppress atopy and autoimmunity. To address the role of environmental factors in type 1 diabetes, large-scale studies are required. For this purpose, the international consortium Environmental Determinants of Diabetes in the Young (TEDDY http www.niddk.nih. gov patient TEDDY TEDDY.htm) has been established so as to follow large number of babies with high-risk HLA genotypes during early life and thus identify...


Eric Gershwin, MD is a distinguished professor of medicine as well as the Jack and Donald Chia professor of medicine. He is also chief of the division of allergy and clinical immunology at the University of California School of Medicine in Davis. Dr. Gershwin graduated from Stanford Medical School in 1971 and subsequently trained in internal medicine and then immunology at Tufts University-New England Medical Center and the National Institutes of Health. He joined the UC Davis faculty in 1975 and has been division chief since 1982. Dr. Gershwin has been continuously funded by NIH since 1975 and currently has published more than 20 books, 600 experimental papers, and 200 book chapters or review articles. He is editor of the Journal of Autoimmunity and Clinical Reviews in Allergy and Immunology and on the editorial board of multiple other journals. His major contributions revolve around the theme of autoimmune disease. Dr. Gershwin was the first individual to clone an autoantigen and...

El Gluten Free Diet

Celiac disease is an inherited autoimmune disease that usually affects several organs in the body before it is diagnosed and treated. When a person with celiac disease consumes any food, beverage, or medication containing wheat, barley, rye, and possibly oats, his or her immune system is triggered and responds by damaging the lining of the intestinal tract. As a result, symptoms include recurrent abdominal pain, bloating, diarrhea, weight loss, lactose intolerance, and malnutrition, often accompanied by nonintestinal symptoms such as anemia and fatigue. Some people have no symptoms whatsoever.

Future directions

Only as nutrients but also as signaling molecules has given rise to the new field of lipidomics, which seeks to profile this vast group. Many of the lipid derivatives are now known to be involved in regulating various stages of cell growth, and the implications extend far beyond wound healing and will likely have significant impact in cancer biology and autoimmune disease. There are several fields that will likely spur advances in wound healing in the next decade. An understanding of the healing process (inflammation, proliferation, and remodeling) in acute and chronic wounds will benefit from genomic, proteomic, and lipidomic profiling of wound tissue and wound fluids. This will undoubtedly lead to the identification of target molecules and pathways for pharmacological intervention to enhance healing, especially in chronic wounds. This profiling will also lead to a better understanding of how nutrients may be used as adjuvants to the healing process. In our laboratory, we have used...


The most important causes of B12 deficiency are the various forms of intestinal malabsorption of which the autoimmune disease, pernicious anaemia, is the most common. Autoantibodies are produced against the parietal cells of the stomach so the cells can no longer produce IF or hydrochloric acid (HCl). Patients with hypochlorhydria, such as the elderly and postgastrectomy patients, may exhibit malabsorption of dietary cobalamin and a lack of IF prevents absorption of vitamin B12.

Deplete DHA

Another event that poses a real health hazard to those receiving vaccines contaminated by animal viruses was discovered by microbiologist Dr. Howard Urnovitz. He found that certain animal viruses could merge genetically with human viruses, producing a hybrid that was fully pathological in humans. These viruses, in an immunologically impaired host, could produce chronic immune responses either as autoimmune diseases or chronic viral infections, causing a lifetime of misery and disability.

Pernicious anemia

Pernicious anemia is an autoimmune disorder caused by production of autoantibodies against the parietal cell antigen, H+ K+-ATPase, and against the parietal cell secretory product, intrinsic factor, resulting in the loss of parietal cells in the fundus and body of the stomach. The loss of these cells is associated with achlorhydria, vitamin B12 deficiency and megaloblastic anemia.58 In children the 'adult form' of pernicious anemia has been reported in association with other autoimmune diseases, such as thyroiditis and diabetes mellitus, and with precancerous and malignant lesions. In childhood, pernicious anemia has been attributed to dietary lack of cobal-amin, to absence of cobalamin in the ileum or to so-called juvenile pernicious anemia. The last is characterized by absent or very low levels of intrinsic factor in the gastric juice, in the absence of achlorhydria and the absence of intrinsic factor or H+ K+-ATPase autoantibodies.58 It has been suggested that this defect could be...

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